Guest Editors
Dr. María Laura Barreiro Arcos
Email: mlbarreiro@yahoo.com.ar
Affiliation: School of Engineering and Agrarian Sciences, Institute of Biomedical Research (BIOMED-UCA-CONICET), Pontifical Catholic University of Argentina, Buenos Aires, C1107AAZ, Argentina
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Research Interests: oxidative stress, redox regulation, mitochondrial dysfunction, bioactive agents, cell signaling, cell death pathways, immune-neuroendocrine diseases, cancer
Assoc. Prof. Horacio Eduardo Romeo
Email: horacio_romeo@uca.edu.ar
Affiliation: School of Engineering and Agrarian Sciences, Institute of Biomedical Research (BIOMED-UCA-CONICET), Pontifical Catholic University of Argentina, Buenos Aires, C1107AAZ, Argentina
Homepage:
Research Interests: euroimmunoendocrinology, microsurgery, neuroanatomy, neuroimmune connection, mucosa-associated lymphoid tissue, nerve-immune crosstalk
Summary
This Special Issue of Biocell invites original research articles, short communications, and review papers that explore the role of inflammation, oxidative stress, and mitochondrial dysfunction in human diseases, and how their modulation can serve as a therapeutic strategy.
Inflammation and oxidative stress are central to the pathophysiology of a wide range of conditions, including cancer, autoimmune disorders, neurodegeneration, and chronic inflammatory diseases. Mitochondria play a critical role in regulating cell fate and energy metabolism and are closely linked to both oxidative damage and inflammatory signaling pathways.
We particularly encourage submissions that address how the positive or negative modulation of these pathways -depending on the pathological context- can contribute to disease prevention, resolution, or therapy. Contributions focusing on natural products, synthetic derivatives, or novel pharmacological agents targeting these mechanisms are welcome.
Topics of interest include (but are not limited to):
• Cellular and molecular mechanisms of inflammation and redox regulation
• Crosstalk between mitochondrial function, oxidative stress, and immune signaling
• Redox imbalance and inflammatory pathways in the pathogenesis of chronic diseases and cancer
• Inflammatory and oxidative mechanisms in neurodegeneration, immune disorders, and metabolic syndromes
• Anatomical substrate for neuroimmunological relationships.
• Natural compounds and synthetic derivatives modulating these pathways
• Pro-oxidant or pro-apoptotic approaches in tumor therapy
• Mitochondrial protective strategies in neurodegenerative and metabolic diseases
• Immunometabolism and inflammation resolution mechanisms
• Antioxidant and anti-inflammatory interventions (in vitro, in vivo, or ex vivo)
Keywords
redox biology, mitochondrial dysfunction, bioactive compounds, cell death pathways, immune-neuroendocrine diseases, metabolic disorders, neurodegenerative diseases, cancer
Published Papers
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